BRAIN-NUTRITION

It is a patented, high-quality form of choline, produced in the USA by Balchem. Choline is considered an essential nutrient because, when the body uses it faster than it can produce it, intake from food or supplements becomes necessary. It may have beneficial effects in supporting attention, learning, and short- and long-term memory. It helps reduce manifestations of acetylcholine deficiency (e.g., panic attacks, brain fog, mild cognitive impairment, mental retardation, Down syndrome, etc.). It participates in the synthesis of phospholipids in the structure of neuronal membranes (phosphatidylcholine, phosphatidylserine, sphingomyelin) and of the neurotransmitter acetylcholine.

It is a non-essential amino acid synthesized in the body under the action of the enzyme glutamine synthetase, mainly found in astrocytes—cells that support the structure and normal functioning of neurons. It helps synthesize and balance the neurotransmitters glutamate (excitatory) and GABA (relaxing), produces ATP energy, reduces excess ammonia, and supports the transmission of nerve impulses. It participates in the glutamate–GABA neurotransmitter synthesis and the glutamate–glutamine cycle, both essential for normal brain activity, and is involved in the development of learning, speech, working memory, and emotional adaptability—including in ADHD and autism spectrum disorders.

Este un aminoacid neesential care poate fi produs in organism din fenilalanina, dar in anumite situatii, precum fenilcetonuria, poate fi necesara suplimentarea. Este un precursor al neurotransmitatorilor norepinefrina, epinefrina si dopamina, contribuind la sustinerea concentrarii, vigilentei mentale, atentiei, starii de spirit, motivatiei si capacitatii de adaptare la stres. Ofera sprijin in reducerea manifestarilor asociate cu ADHD, depresie, tulburare bipolara (faza depresiva), sindromul picioarelor nelinistite, dependente, sevraj, tulburare opozitionala si fenilcetonurie, si creste capacitatea organismului de a se adapta la stresul cronic prin reglarea axei hipotalamo-hipofizo-suprarenale (HPA).

It is a precursor of NAD+ (nicotinamide adenine dinucleotide, oxidized form) and helps reduce tiredness and fatigue by maintaining normal energy balance. It supports cellular energy production through redox reactions and may help restore the optimal NAD+/NADH ratio. It protects cells against oxidative stress, reduces brain inflammation, and supports cell-to-cell communication. Additionally, it helps with axon remyelination and supports dopamine synthesis.

It supports the synthesis of the neurotransmitters serotonin, dopamine, GABA, norepinephrine, and epinephrine, as well as synaptic plasticity and myelin formation. It helps reduce manifestations of depression, vitamin B6-deficiency-related epilepsy, autism spectrum disorders, ADHD, and cognitive decline. It acts as a cofactor in the conversion of tyrosine to dopamine, dopamine to norepinephrine, tryptophan to serotonin, and glutamate to GABA (gamma-aminobutyric acid). It supports brain neuroplasticity, enhances cognitive function, and reduces mental fatigue by improving brain oxygenation.

It has high absorption and is well tolerated by children. It helps protect neurons against oxidative stress and inflammatory processes by reducing NF-κB activity. It has beneficial effects in supporting focus, memory, and sleep quality, and helps reduce migraines, chronic pain, epilepsy, depression and anxiety, ADHD, autism spectrum disorders, oppositional defiant disorder, and brain fog. It protects cells against oxidative stress and reduces inflammation, while supporting the integrity of the blood-brain barrier, which protects the brain. It supports nerve impulse transmission by maintaining myelin structure, reduces glutamate toxicity by inhibiting NMDA receptors, and prevents excessive calcium accumulation in neurons by moderately blocking voltage-gated calcium channels. In addition, it helps maintain electrolyte balance.

It is the bioactive form of inositol that acts in the brain and may have beneficial effects in reducing manifestations of depression, obsessive-compulsive disorder, panic attacks, anxiety, bulimia, bipolar disorder (depressive phase), mood swings, and ADHD. It exerts these effects by participating in the synthesis of the neurotransmitters serotonin and dopamine, improving the efficiency of serotonin and dopamine by increasing their receptor density, helping transmit nerve impulses by enhancing synaptic plasticity, and balancing emotional states and the behavioral manifestations associated with them. Additionally, it protects cells against oxidative stress by activating the nuclear factor erythroid 2 (NRF2).